Reactome: A Curated Pathway Database

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Pathways authored by Wu, J (180291)

DB_ID Name
380972 Energy dependent regulation of mTOR by LKB1-AMPK

Details on Person Wu, J

Class:IdPerson:180291
_displayNameWu, J
_timestamp2017-08-22 20:38:21
created[InstanceEdit:180315] Jassal, B, 2006-05-18 13:16:38
initialJ
surnameWu
(author)[LiteratureReference:180316] Phosphotyrosines 627 and 659 of Gab1 constitute a bisphosphoryl tyrosine-based activation motif (BTAM) conferring binding and activation of SHP2
[LiteratureReference:180502] Roles of Gab1 and SHP2 in paxillin tyrosine dephosphorylation and Src activation in response to epidermal growth factor
[LiteratureReference:199381] TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling
[LiteratureReference:210108] Immunoreceptor DAP12 bearing a tyrosine-based activation motif is involved in activating NK cells
[LiteratureReference:400652] Solution structure of the first intracellular loop of prostacyclin receptor and implication of its interaction with the C-terminal segment of G alpha s protein
[LiteratureReference:417791] Netrin requires focal adhesion kinase and Src family kinases for axon outgrowth and attraction
[LiteratureReference:418679] Netrin signal transduction and the guanine nucleotide exchange factor DOCK180 in attractive signaling
[LiteratureReference:428076] Loss of signaling through the G protein, Gz, results in abnormal platelet activation and altered responses to psychoactive drugs
[LiteratureReference:430166] Vav and SLP-76 interact and functionally cooperate in IL-2 gene activation
[LiteratureReference:444875] INSL5 is a high affinity specific agonist for GPCR142 (GPR100)
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