Reactome: A Curated Pathway Database

Query author contributions in Reactome

Reactome depends on collaboration between our curation team and outside experts to assemble and peer-review its pathway modules. The integration of ORCID within Reactome enables us to meet a key challenge with authoring, curating and reviewing biological information by incentivizing and crediting the external experts that contribute their expertise and time to the Reactome curation process. More information is available at ORCID and Reactome.

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Pathways reviewed by Dixit, VM (141149)

DB_ID Name
349425 Autodegradation of the E3 ubiquitin ligase COP1

Details on Person Dixit, VM

_displayNameDixit, VM
_timestamp2017-08-22 20:37:39
created[InstanceEdit:141150] Gillespie, ME, 2004-09-02 19:17:31
crossReference[DatabaseIdentifier:5625820] ORCID:0000-0001-6983-0326
modified[InstanceEdit:5625812] Matthews, Lisa, 2014-10-07
(author)[LiteratureReference:114472] ML-IAP, a novel inhibitor of apoptosis that is preferentially expressed in human melanomas.
[LiteratureReference:140483] FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis
[LiteratureReference:141158] Death receptors: signaling and modulation
[LiteratureReference:349437] ATM engages autodegradation of the E3 ubiquitin ligase COP1 after DNA damage
[LiteratureReference:352258] The CED-3/ICE-like protease Mch2 is activated during apoptosis and cleaves the death substrate lamin A
[LiteratureReference:450119] IRAK (Pelle) family member IRAK-2 and MyD88 as proximal mediators of IL-1 signaling
[LiteratureReference:688977] Transcriptional activation of the tumor necrosis factor alpha-inducible zinc finger protein, A20, is mediated by kappa B elements
[LiteratureReference:844450] Inflammasomes: guardians of cytosolic sanctity
[LiteratureReference:873998] Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf
[LiteratureReference:874008] The Birc1e cytosolic pattern-recognition receptor contributes to the detection and control of Legionella pneumophila infection
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